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Additional resources for A monograph of the Ithomiidae (Lepidoptera) Part IV: The tribe Napeogenini Fox, (American Entomological Institute. Memoir)

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Consequently, our program will compare tumor viruses with closely related non-tumor viruses. The long-term objective of the program is to better understand the mechanisms by which herpesviruses persist and contribute to oncogenesis in the infected host. We will search for additional viral genes that mediate persistence and oncogenicity, and we will study the mechanism of action of new genes that are identified. We also will identify cellular genes whose level of expression change after infection, and test the hypothesis that some of these altered cellular genes influence the outcome of the virus-host interaction, contributing to the persistence and/or oncogenicity of the viruses.

While the LAT gene is known to be the only gone that is abundantly transcribed during latency, it does not appear to code for any known protein. 5 kb LAT transcript. This proposal aims to dissect out the transcriptional control of the LAT promoter and its associated enhancer region from three distinct strains of HSV-1. These three strains of HSV-1 differ in their ability to undergo spontaneous and stress-induced reactivation. If the pathway by which the virus senses and responds to adrenergic stimuli can be established, it should prove to be a rich source of therapeutic targets.

Professor; Massachusetts General Hospital 55 Fruit St Boston, Ma 02114 Timing: Fiscal Year 2001; Project Start 23-JAN-1987; Project End 31-AUG-2006 Summary: (provided by applicant): Studies will be undertaken to develop modes of gene delivery to experimental neural tumors for therapeutic intervention. Neoplastic lesions associated with tuberous sclerosis (TSC) including subependymal glial nodules, giant cell astrocytomas and cortical hamartomas, are believed to represent the consequences of loss of tumor suppressor genes on growth of astrocytes, neuroprogenitor cells and mesenchymal elements.

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